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Drawing on the hallmarks of aging framework, the authors examine the contributions of chronic low-grade inflammation, or inflammaging, immunosenescence, mitochondrial dysfunction, cellular senescence, altered nutrient sensing, and impaired stress resilience
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Studies have demonstrated that DSIP reduces markers of oxidative damage — including lipid peroxidation and protein oxidation — in brain tissue, and upregulates endogenous antioxidant defence enzymes including superoxide dismutase (SOD), catalase, and glutathione peroxidase
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