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Thus, GHRP-6 represents a potential therapeutic agent to counteract muscle toxicity associated with DOX, acting through mechanisms that integrate autophagy, survival signaling, growth factor preservation, and oxidative stress regulation [2]

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In particular, its ability to contribute to acetylation of -OH or -NH2 functional groups on amino acids and N-terminal groups in proteins [2,7,8,50,51] has opened up new avenues to explain mechanisms underlying important LAC activities