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Key findings include: Decreased sensitivity of neuronal receptors to excitatory glutamate stimulation Attenuation of excitotoxic processes, which may protect neurons under ischemic or stress conditions NMDA interaction proposed as a primary CNS mechanism in multiple in vitro studies Potential role in reducing calcium-mediated neuronal damage during hypoxia GABAergic System Interactions – Inhibitory Neurotransmission Studies have documented DSIP’s enhancement of GABA-activated currents in hippocampal and cerebellar neuron preparations[2]